Prenatal administration of retinoic acid upregulates insulin-like growth factor receptors in the nitrofen-induced hypoplastic lung
Author:
Publisher
Wiley
Subject
Health, Toxicology and Mutagenesis,Developmental Biology,Toxicology,Embryology
Reference29 articles.
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3. Prenatal retinoic acid up-regulates pulmonary gene expression of Coup-TFII, Fog2, and GATA4 in pulmonary hypoplasia;Doi;J Pediatr Surg,2009
4. Congenital diaphragmatic hernia: a retinoid signaling pathway disruption during lung development?;Gallot;Birth Defects Res A Clin Mol Teratol,2005
5. High glucose and insulin decrease fetal lung insulin receptor mRNA and tyrosine kinase activity in vitro;Gewolb;Biochem Biophys Res Commun,1994
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1. Disruptions in retinoic acid signaling pathway contribute to abnormal lung development in congenital diaphragmatic hernia: a therapeutic potential for retinoids to attenuate pulmonary hypoplasia;Pediatric Research;2024-02-10
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3. Congenital Diaphragmatic Hernia: State of the Art in Translating Experimental Research to the Bedside;European Journal of Pediatric Surgery;2019-07-31
4. Assessment of the nitrofen model of congenital diaphragmatic hernia and of the dysregulated factors involved in pulmonary hypoplasia;Pediatric Surgery International;2018-11-01
5. Lung function and pulmonary artery blood flow following prenatal maternal retinoic acid and imatinib in the nitrofen model of congenital diaphragmatic hernia;Journal of Pediatric Surgery;2018-09
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