The dual role of boron in vitro neurotoxication of glioblastoma cells via <scp>SEMA3F</scp> / <scp>NRP2</scp> and ferroptosis signaling pathways-Reference-Cited by-同舟云学术

The dual role of boron in vitro neurotoxication of glioblastoma cells via SEMA3F / NRP2 and ferroptosis signaling pathways

Published:2022-09-22 Issue:1 Volume:38 Page:70-77
ISSN:1520-4081
Container-title:Environmental Toxicology
language:en
Short-container-title:Environmental Toxicology

Author:

Kar Fatih¹^ORCID,Hacioğlu Ceyhan²^ORCID,Kaçar Sedat³⁴^ORCID

Affiliation:

1. Department of Basic Science, Faculty of Engineering and Natural Science Kutahya Health Sciences University Kutahya Turkey

2. Department of Biochemistry, Faculty of Pharmacy Duzce University Duzce Turkey

3. Department of Histology and Embryology, Faculty of Medicine Eskisehir Osmangazi University Eskisehir Turkey

4. Department of Surgery Indiana University School of Medicine, Indiana University Health Comprehensive Wound Center, Indiana Center for Regenerative Medicine and Engineering Indianapolis IN USA

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Management, Monitoring, Policy and Law,Toxicology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/tox.23662

Reference31 articles.

1. Iron‐induced cellular in vitro neurotoxic responses in ratC6cell line

2. Biology of Glioblastoma Multiforme—Exploration of Mitotic Catastrophe as a Potential Treatment Modality

3. Novel Treatment Strategies for Glioblastoma

4. Concanavalin A induces apoptosis in a dose‐dependent manner by modulating thiol/disulfide homeostasis in C6 glioblastoma cells

5. Harnessing oxidative stress for anti-glioma therapy

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