Progressive disruption of neurodevelopment by mid‐gestation exposure to lipopolysaccharides and the ameliorating effect of continuous alpha‐glycosyl isoquercitrin treatment-Reference-Cited by-同舟云学术

Progressive disruption of neurodevelopment by mid‐gestation exposure to lipopolysaccharides and the ameliorating effect of continuous alpha‐glycosyl isoquercitrin treatment

Published:2022-09-20 Issue:1 Volume:38 Page:49-69
ISSN:1520-4081
Container-title:Environmental Toxicology
language:en
Short-container-title:Environmental Toxicology

Author:

Okano Hiromu¹²,Takashima Kazumi¹²,Takahashi Yasunori¹²,Ojiro Ryota¹²,Tang Qian¹²,Ozawa Shunsuke¹²^ORCID,Zou Xinyu¹²,Koyanagi Mihoko³,Maronpot Robert R.⁴^ORCID,Yoshida Toshinori¹²^ORCID,Shibutani Makoto¹²⁵^ORCID

Affiliation:

1. Laboratory of Veterinary Pathology Tokyo University of Agriculture and Technology Tokyo Japan

2. Cooperative Division of Veterinary Sciences, Graduate School of Agriculture Tokyo University of Agriculture and Technology Tokyo Japan

3. Global Scientific and Regulatory Affairs San‐Ei Gen F.F.I. Inc. Osaka Japan

4. Maronpot Consulting, LLC Raleigh North Carolina USA

5. Institute of Global Innovation Research, Tokyo University of Agriculture and Technology Tokyo Japan

Funder

Institute of Global Innovation Research, Tokyo University of Agriculture and Technology

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Management, Monitoring, Policy and Law,Toxicology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/tox.23661

Reference66 articles.

1. Effects of prenatal infection on brain development and behavior: A review of findings from animal models

2. Maternal immune activation: Implications for neuropsychiatric disorders

3. Controversies and prospects about microglia in maternal immune activation models for neurodevelopmental disorders

4. Corynoline protects lipopolysaccharide‐induced mastitis through regulating AKT / GSK3β / Nrf2 signaling pathway

5. Alpinae Oxyphyllae Fructus alleviated LPS ‐induced cognitive impairments via PI3K / AKT / NF‐κB signaling pathway

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