PI-3K/Akt signal pathway plays a crucial role in arsenite-induced cell proliferation of human keratinocytes through induction of cyclin D1
Author:
Publisher
Wiley
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference50 articles.
1. 3-Phosphoinositide-dependent protein kinase-1 (PDK1): structural and functional homology with the Drosophila DSTPK61 kinase
2. Transcription-dependent and -independent control of neuronal survival by the PI3K–Akt signaling pathway
3. Arsenic-induced enhancement of ultraviolet radiation carcinogenesis in mouse skin: a dose-response study.
4. The Phosphoinositide 3-Kinase Pathway
5. The phosphoinositide 3-kinase/Akt pathway regulates cell cycle progression of HL60 human leukemia cells through cytoplasmic relocalization of the cyclin-dependent kinase inhibitor p27Kip1 and control of cyclin D1 expression
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2. Low-dose arsenic trioxide enhances membrane-GLUT1 expression and glucose uptake via AKT activation to support L-02 cell aberrant proliferation;Toxicology;2022-06
3. Malignant growth of arsenic-transformed cells depends on activated Akt induced by reactive oxygen species;International Journal of Environmental Health Research;2022-01-02
4. Arsenic exposure: A public health problem leading to several cancers;Regulatory Toxicology and Pharmacology;2020-02
5. Akt Regulated Phosphorylation of GSK-3β/Cyclin D1, p21 and p27 Contributes to Cell Proliferation Through Cell Cycle Progression From G1 to S/G2M Phase in Low-Dose Arsenite Exposed HaCat Cells;Frontiers in Pharmacology;2019-10-11
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