Brief Report: Loss of p15Ink4b Accelerates Development of Myeloid Neoplasms in Nup98‐HoxD13 Transgenic Mice

Author:

Humeniuk Rita1,Koller Richard1,Bies Juraj1,Aplan Peter1,Wolff Linda1

Affiliation:

1. National Cancer Institute, National Institutes of HealthBethesda Maryland USA

Publisher

Oxford University Press (OUP)

Subject

Cell Biology,Developmental Biology,Molecular Medicine

Reference17 articles.

1. Incidence of the myelodysplastic syndromes using a novel claims-based algorithm: high number of uncaptured cases by cancer registries

2. Review of alterations of the cyclin-dependent kinase inhibitor INK4 family genes p15, p16, p18 and p19 in human leukemia–lymphoma cells

3. Distinct patterns of inactivation of p15INK4B and p16INK4A characterize the major types of hematological malignancies;Herman JG;Cancer Res,1997

4. Hypermethylation‐associated inactivation indicates a tumor suppressor role for p15INK4B;Herman JG;Cancer Res,1996

5. Methylation-Independent Silencing of the Tumor Suppressor INK4b (p15) by CBFβ-SMMHC in Acute Myelogenous Leukemia with inv(16)

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