Cystatin B increases autophagic flux by sustaining proteolytic activity of cathepsin B and fuels glycolysis in pancreatic cancer: CSTB orchestrates autophagy and glycolysis in PDAC-Reference-Cited by-同舟云学术

Cystatin B increases autophagic flux by sustaining proteolytic activity of cathepsin B and fuels glycolysis in pancreatic cancer: CSTB orchestrates autophagy and glycolysis in PDAC

Published:2022-12 Issue:12 Volume:12 Page:
ISSN:2001-1326
Container-title:Clinical and Translational Medicine
language:en
Short-container-title:Clinical & Translational Med

Author:

Jiang Yongsheng¹²³,Han Lijie¹²³,Xue Meilin¹²³,Wang Ting⁴,Zhu Youwei¹²³,Xiong Cheng¹²³,Shi Minmin¹²³,Li Hongzhe¹²³,Hai Wangxi⁵,Huo Yanmiao⁶,Shen Baiyong¹²³⁷,Jiang Lingxi¹²³,Chen Hao¹²³⁷

Affiliation:

1. Department of General Surgery Pancreatic Disease Center Ruijin Hospital Shanghai Jiao Tong University School of Medicine Shanghai China

2. Research Institute of Pancreatic Diseases Shanghai Jiao Tong University School of Medicine Shanghai China

3. State Key Laboratory of Oncogenes and Related Genes Shanghai Jiao Tong University School of Medicine Shanghai China

4. Department of Pathology Ruijin Hospital Shanghai Jiao Tong University School of Medicine Shanghai China

5. Department of Nuclear Medicine Ruijin Hospital Shanghai Jiao Tong University School of Medicine Shanghai China

6. Department of Biliary‐Pancreatic Surgery Renji Hospital School of Medicine Shanghai Jiao Tong University Shanghai P. R. China

7. Institute of Translational Medicine Shanghai Jiao Tong University Shanghai China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Molecular Medicine,Medicine (miscellaneous)

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ctm2.1126

Reference63 articles.

1. Autophagy and Tumor Metabolism

2. Autophagy—a key player in cellular and body metabolism

3. Targeting autophagy in cancer

4. Autophagy: Renovation of Cells and Tissues

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