Induction of embryonic dysmorphogenesis by high glucose concentration, disturbed inositol metabolism, and inhibited protein kinase C activity-Reference-Cited by-同舟云学术

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Induction of embryonic dysmorphogenesis by high glucose concentration, disturbed inositol metabolism, and inhibited protein kinase C activity

Published:2001-04-19 Issue:5 Volume:63 Page:193-201
ISSN:0040-3709
Container-title:Teratology
language:en
Short-container-title:Teratology

Author:

Wentzel Parri,Wentzel Christian R.,Gäreskog Mattias B.,Eriksson Ulf J.

Funder

Ernfors Family Fund

Söderberg Foundation

Swedish Diabetes Association

Novo Nordisk Foundation

Swedish Medical Research Council

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Developmental Biology,Toxicology,Embryology

Reference55 articles.

1. The relation between thromboxane and prostaglandin synthesis in human decidua tissue: a comparison of eicosanoid synthesis in minced tissue with that in a cell-free preparation;Asboth;Biochim Biophys Acta,1989

2. Altered expression and subcellular localization of diacylglycerol-sensitive protein kinase C isoforms in diabetic rat glomerular cells;Babazono;Diabetes,1998

3. Myo-inositol and prostaglandins reverse the glucose inhibition of neural tube fusion in cultured mouse embryos;Baker;Diabetologia,1990

4. Alpha, beta I, beta II, delta, and epsilon protein kinase C isoforms and compound activity in the sciatic nerve of normal and diabetic rats;Borghini;J Neurochem,1994

5. Outcomes of pregnancy in insulin dependent diabetic women: results of a five year population cohort study;Casson;BMJ,1997

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