Cell surface IL‐1α trafficking is specifically inhibited by interferon‐γ, and associates with the membrane via IL‐1R2 and GPI anchors-Reference-Cited by-同舟云学术

Cell surface IL‐1α trafficking is specifically inhibited by interferon‐γ, and associates with the membrane via IL‐1R2 and GPI anchors

Published:2020-06-08 Issue:11 Volume:50 Page:1663-1675
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Chan Julie N.E.¹,Humphry Melanie¹,Kitt Lauren¹,Krzyzanska Dominika¹,Filbey Kara J.²,Bennett Martin R.¹,Clarke Murray C.H.¹^ORCID

Affiliation:

1. Division of Cardiovascular Medicine Department of Medicine University of Cambridge Addenbrooke's Hospital Cambridge UK

2. Manchester Collaborative Centre for Inflammation Research Lydia Becker Institute of Immunology and Inflammation The University of Manchester Core Technology Facility Manchester UK

Funder

British Heart Foundation

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/eji.201948521

Reference44 articles.

1. Immunological and Inflammatory Functions of the Interleukin-1 Family

2. Interleukin receptor activates a MYD88–ARNO–ARF6 cascade to disrupt vascular stability

3. The IL-1 family: regulators of immunity

4. Cell surface-bound IL-1 is an upstream regulator of the senescence-associated IL-6/IL-8 cytokine network

5. Senescent Vascular Smooth Muscle Cells Drive Inflammation Through an Interleukin-1α–Dependent Senescence-Associated Secretory Phenotype

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