Nrf2 expression driven by Foxp3 specific deletion of Keap1 results in loss of immune tolerance in mice-Reference-Cited by-同舟云学术

Nrf2 expression driven by Foxp3 specific deletion of Keap1 results in loss of immune tolerance in mice

Published:2020-01-14 Issue:4 Volume:50 Page:515-524
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Klemm Patricia¹,Rajendiran Anandhi¹,Fragoulis Athanassios²,Wruck Christoph²,Schippers Angela¹,Wagner Norbert¹,Bopp Tobias³,Tenbrock Klaus¹,Ohl Kim¹^ORCID

Affiliation:

1. Department of Pediatrics Medical Faculty RWTH Aachen Aachen Germany

2. Department of Anatomy and Cell Biology Medical Faculty RWTH Aachen Aachen Germany

3. Institute of Immunology University Medical Center The Johannes Gutenberg University Mainz Mainz Germany

Funder

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/eji.201948285

Reference28 articles.

1. The Nrf2-Antioxidant Response Element Signaling Pathway and Its Activation by Oxidative Stress

2. Distinct Cysteine Residues in Keap1 Are Required for Keap1-Dependent Ubiquitination of Nrf2 and for Stabilization of Nrf2 by Chemopreventive Agents and Oxidative Stress

3. Cell Survival Responses to Environmental Stresses Via the Keap1-Nrf2-ARE Pathway

4. Nrf2 in health and disease: current and future clinical implications

5. Multiorgan Autoimmune Inflammation, Enhanced Lymphoproliferation, and Impaired Homeostasis of Reactive Oxygen Species in Mice Lacking the Antioxidant-Activated Transcription Factor Nrf2

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