Paeoniflorin inhibited nod‐like receptor protein‐3 inflammasome and NF‐κB ‐mediated inflammatory reactions in diabetic foot ulcer by inhibiting the chemokine receptor CXCR2-Reference-Cited by-同舟云学术

Paeoniflorin inhibited nod‐like receptor protein‐3 inflammasome and NF‐κB ‐mediated inflammatory reactions in diabetic foot ulcer by inhibiting the chemokine receptor CXCR2

Published:2020-11-24 Issue:3 Volume:82 Page:404-411
ISSN:0272-4391
Container-title:Drug Development Research
language:en
Short-container-title:Drug Dev Res

Author:

Sun Xiaolong¹^ORCID,Wang Xu¹,Zhao Zhenyu¹,Chen Jing¹,Li Cheng¹,Zhao Gang²

Affiliation:

1. The Second Department of Surgery, First Affiliated Hospital Heilongjiang University of Chinese Medicine Harbin Heilongjiang China

2. Department of Peripheral Vascular Disease, First Affiliated Hospital Heilongjiang University of Chinese Medicine Harbin Heilongjiang China

Publisher

Wiley

Subject

Drug Discovery

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ddr.21763

Reference31 articles.

1. Diabetes causes marked changes in function and metabolism of rat neutrophils;Alba‐Loureiro T. C.;The Journal of Endocrinology,2006

2. Bone marrow mesenchymal stem cell aggregate: an optimal cell therapy for full-layer cutaneous wound vascularization and regeneration

3. Diabetic Foot Ulcers and Their Recurrence

4. Chemokines in pathology and medicine

5. CXCL1 and CXCL2 Regulate NLRP3 Inflammasome Activation via G-Protein–Coupled Receptor CXCR2

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