Maternal antioxidant supplementation does not reduce the incidence of phenytoin-induced cleft lip and related malformations in rats
Author:
Publisher
Wiley
Subject
Health, Toxicology and Mutagenesis,Developmental Biology,Toxicology,Embryology
Reference37 articles.
1. 2003. Phenytoin teratogenesis: drug-induced embryonic hypoxia. Honour Thesis, University of Sydney: Sydney, Australia. p 1-99.
2. The contribution of maternal epilepsy and its treatment to the etiology of oral clefts: A population based case-control study
3. Pharmacologically induced embryonic dysrhythmia and episodes of hypoxia followed by reoxygenation: A common teratogenic mechanism for antiepileptic drugs?
4. Phenytoin-induced cleft palate: Evidence for embryonic cardiac bradyarrhythmia due to inhibition of delayed rectifier k+ channels resulting in hypoxia-reoxygenation damage
5. Embryonic Arrhythmia by Inhibition of HERG Channels: A Common Hypoxia-related Teratogenic Mechanism for Antiepileptic Drugs?
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1. The effect of phenytoin on embryonic heart rate in Vivo;Reproductive Toxicology;2021-12
2. Fetal hypoxia and hyperglycemia in the formation of phenytoin‐induced cleft lip and maxillary hypoplasia;Epilepsia Open;2019-07-29
3. The Effect of Dofetilide on the Heart Rate of GD11 and GD13 Rat Embryos, in vivo, Using Ultrasound;Birth Defects Research Part B: Developmental and Reproductive Toxicology;2015-09-24
4. A comparison of drug-induced cardiotoxicity in rat embryos cultured in human serum or protein free media;Journal of Pharmacological and Toxicological Methods;2014-11
5. Antiepileptic drugs and pregnancy outcomes;American Journal of Medical Genetics Part A;2012-06-18
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