Neuronal migration is transiently delayed by prenatal exposure to intermittent hypoxia-Reference-Cited by-同舟云学术

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Neuronal migration is transiently delayed by prenatal exposure to intermittent hypoxia

Published:2005 Issue:4 Volume:74 Page:287-299
ISSN:1542-9733
Container-title:Birth Defects Research Part B: Developmental and Reproductive Toxicology
language:en
Short-container-title:Birth Defect Res B

Author:

Zechel Jennifer L.,Gamboa Jorge L.,Peterson Allyn G.,Puchowicz Michelle A.,Selman Warren R.,Lust W. David

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Developmental Biology,Toxicology,Embryology

Reference57 articles.

1. The balance between copper-zinc superoxide dismutase and catalase affects the sensitivity of mouse epidermal cells to oxidative stress

2. . 1991. Neocortical development. New York: Raven Press.

3. Delayed neuronal migration of protein kinase Cγ immunoreactive cells in hippocampal CA1 area after 48 h of moderate hypoxemia in the near term ovine fetus

4. Catalase Activity Is Regulated by c-Abl and Arg in the Oxidative Stress Response

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2. Maternal Hyperhomocysteinemia Disturbs the Mechanisms of Embryonic Brain Development and Its Maturation in Early Postnatal Ontogenesis;Cells;2023-01-03

3. Maternal Hypoxia Increases the Excitability of Neurons in the Entorhinal Cortex and Dorsal Hippocampus of Rat Offspring;Frontiers in Neuroscience;2022-04-12

4. Intermittent Hypoxia Impairs Trophoblast Cell Viability by Triggering the Endoplasmic Reticulum Stress Pathway;Reproductive Sciences;2020-02

5. Prenatal Environment That Affects Neuronal Migration;Frontiers in Cell and Developmental Biology;2019-07-17

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