Retracted : Knockdown of cathepsin B and u PAR inhibits CD 151 and α3β1 integrin‐mediated cell adhesion and invasion in glioma
Author:
Affiliation:
1. Department of Cancer Biology and PharmacologyCollege of Medicine at Peoria, University of IllinoisPeoriaIL
2. Department of NeurosurgeryCollege of Medicine at Peoria, University of IllinoisPeoriaIL
Publisher
Wiley
Subject
Cancer Research,Molecular Biology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/mc.21915
Reference43 articles.
1. Integrin α3β1-mediated interaction with laminin-5 stimulates adhesion, migration and invasion of malignant glioma cells
2. RNAi-mediated inhibition of cathepsin B and uPAR leads to decreased cell invasion, angiogenesis and tumor growth in gliomas
3. Regulation of Urokinase Receptor Proteolytic Function by the Tetraspanin CD82
4. The fibrinolytic receptor for urokinase activates the G protein-coupled chemotactic receptor FPRL1/LXA4R
5. Urokinase Receptors Promote β1 Integrin Function through Interactions with Integrin α3β1
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