Glutamate potentiates heterologously expressed homomeric acid‐sensing ion channel 1a
Author:
Affiliation:
1. Laboratory of Biophysics of Synaptic Processes Sechenov Institute of Evolutionary Physiology and Biochemistry RAS St. Petersburg Russia
Funder
Russian Science Foundation
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/syn.22227
Reference21 articles.
1. Heteroarylguanidines as Allosteric Modulators of ASIC1a and ASIC3 Channels
2. Subtype-specific Modulation of Acid-sensing Ion Channel (ASIC) Function by 2-Guanidine-4-methylquinazoline
3. Alternative Splicing and Interaction with Di- and Polyvalent Cations Control the Dynamic Range of Acid-sensing Ion Channel 1 (ASIC1)
4. Pharmacology of acid-sensing ion channels – Physiological and therapeutical perspectives
5. The function and regulation of acid‐sensing ion channels (ASICs) and the epithelial Na + channel (ENaC): IUPHAR Review 19
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1. Glutamate acts on acid-sensing ion channels to worsen ischaemic brain injury;Nature;2024-07-10
2. Reduced Plasma-Membrane Calcium ATPase Activity and Extracellular Acidification Trigger Presynaptic Homeostatic Potentiation at the Mouse Neuromuscular Junction;Neuroscience;2023-11
3. Possible Compensatory Role of ASICs in Glutamatergic Synapses;International Journal of Molecular Sciences;2023-08-19
4. Group II metabotropic glutamate receptor activation attenuates acid-sensing ion channel currents in rat primary sensory neurons;Journal of Biological Chemistry;2023-03
5. Increased seizure sensitivity in pregnant mice with genetic knockdown of acid sensing ion channel 2a is associated with impaired hippocampal inflammatory response;Frontiers in Physiology;2022-09-14
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