LncRNA MEG3 mediates nickel oxide nanoparticles‐induced pulmonary fibrosis via suppressing TGF ‐β1 expression and epithelial‐mesenchymal transition process-Reference-Cited by-同舟云学术

LncRNA MEG3 mediates nickel oxide nanoparticles‐induced pulmonary fibrosis via suppressing TGF ‐β1 expression and epithelial‐mesenchymal transition process

Published:2021-02-06 Issue:6 Volume:36 Page:1099-1110
ISSN:1520-4081
Container-title:Environmental Toxicology
language:en
Short-container-title:Environmental Toxicology

Author:

Zhan Haibing¹,Chang Xuhong¹,Wang Xiaoxia¹,Yang Mengmeng¹,Gao Qing¹,Liu Han¹,Li Chengyun¹,Li Sheng²,Sun Yingbiao¹^ORCID

Affiliation:

1. Department of Toxicology, School of Public Health Lanzhou University Lanzhou China

2. Department of Public Health The First People's Hospital of Lanzhou City Lanzhou China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Management, Monitoring, Policy and Law,Toxicology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/tox.23109

Reference56 articles.

1. Nickel Carcinogenesis Mechanism: DNA Damage

2. Distinct mechanisms of oxidative DNA damage induced by carcinogenic nickel subsulfide and nickel oxides.

3. Advancing risk assessment of engineered nanomaterials: Application of computational approaches

4. Mesoporous nickel oxide nanostructures: influences of crystalline defects and morphological features on mediator-free electrochemical monosaccharide sensor application

5. Effects of physicochemical properties of nanomaterials on their toxicity

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