A novel mechanism of Smads/miR‐675/TGFβR1 axis modulating the proliferation and remodeling of mouse cardiac fibroblasts-Reference-Cited by-同舟云学术

A novel mechanism of Smads/miR‐675/TGFβR1 axis modulating the proliferation and remodeling of mouse cardiac fibroblasts

Published:2019-04-05 Issue:11 Volume:234 Page:20275-20285
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Wang Lei¹,Jiang Ping²,He Yi²,Hu Hongyu²,Guo Yuan²,Liu Xiangyang²,Qiu Haihua²,Ma Qilin¹,Ouyang Fan²^ORCID

Affiliation:

1. Department of Cardiology Xiangya Hospital, Central South University Changsha Hunan China

2. Department of Cardiology Zhuzhou Central Hospital, the Affiliated Zhuzhou Hospital of Xiangya Medical College of Central South University Zhuzhou Hunan China

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.28628

Reference44 articles.

1. Regulation of Myocardial Fibrosis by MicroRNAs

2. Therapeutic inhibition of the miR-34 family attenuates pathological cardiac remodeling and improves heart function

3. miR-107 orchestrates ER stress induction and lipid accumulation by post-transcriptional regulation of fatty acid synthase in hepatocytes

4. Myocardial Fibrosis in Transforming Growth Factor β1Heterozygous Mice

5. Crosstalk between TGF-β signaling and the microRNA machinery

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