Honokiol attenuate the arsenic trioxide‐induced cardiotoxicity by reducing the myocardial apoptosis-Reference-Cited by-同舟云学术

Honokiol attenuate the arsenic trioxide‐induced cardiotoxicity by reducing the myocardial apoptosis

Published:2022-02-16 Issue:2 Volume:10 Page:
ISSN:2052-1707
Container-title:Pharmacology Research & Perspectives
language:en
Short-container-title:Pharmacology Res & Perspec

Author:

Huang An‐Liang¹²,Yang Fan¹²^ORCID,Cheng Ping³,Liao Dian‐ying⁴,Zhou Li¹²,Ji Xing‐Li¹²,Peng Dou‐Dou¹²,Zhang Li¹²,Cheng Ting‐Ting¹²,Ma Li¹²,Xia Xian‐Gen¹²

Affiliation:

1. Department of Pathology Chengdu Fifth People’s Hospital Chengdu Sichuan People’s Republic of China

2. Department of Pathology The Fifth Affiliated People's Hospital of Chengdu University of Traditional Chinese Medicine Chengdu Sichuan People’s Republic of China

3. State Key Lab of Biotherapy West China Hospital Sichuan University Chengdu Sichuan People’s Republic of China

4. Department of Pathology West China Hospital Chengdu Sichuan People’s Republic of China

Publisher

Wiley

Subject

General Pharmacology, Toxicology and Pharmaceutics,Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/prp2.914

Reference44 articles.

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2. Arsenic trioxide induces apoptosis in NB-4, an acute promyelocytic leukemia cell line, through up-regulation of p73 via suppression of nuclear factor kappa B-mediated inhibition of p73 transcription and prevention of NF-κB-mediated induction of XIAP, cIAP2, BCL-XL and survivin

3. Alteration in miRNA gene expression pattern in acute promyelocytic leukemia cell induced by arsenic trioxide: a possible mechanism to explain arsenic multi-target action