Cellular stress and apoptosis contribute to the pathogenesis of autism spectrum disorder

Author:

Dong Daoyin1,Zielke Horst Ronald2,Yeh David1,Yang Peixin13ORCID

Affiliation:

1. Department of Obstetrics; Gynecology & Reproductive Sciences, University of Maryland School of Medicine; Baltimore Maryland

2. Department of Pediatrics; University of Maryland School of Medicine; Baltimore Maryland

3. Department of Biochemistry and Molecular Biology; University of Maryland School of Medicine; Baltimore Maryland

Funder

NIH

Publisher

Wiley

Subject

Genetics(clinical),Clinical Neurology,General Neuroscience

Reference69 articles.

1. Prevalence of autism spectrum disorders-Autism and Developmental Disabilities Monitoring Network, 14 sites, United States, 2008;Morbidity and Mortality Weekly Report. Surveillance Summaries,2012

2. Endoplasmic reticulum stress and IRE-1 signaling cause apoptosis in colon cancer cells in response to andrographolide treatment;Banerjee;Oncotarget,2016

3. Neuroanatomic observations of the brain in autism: A review and future directions;Bauman;International Journal of Developmental Neuroscience,2005

4. NO signaling in the CNS: From the physiological to the pathological;Bishop;Toxicology,2005

5. Postnatal exposure to trichloroethylene alters glutathione redox homeostasis, methylation potential, and neurotrophin expression in the mouse hippocampus;Blossom;Neurotoxicology,2012

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