Synaptic structural abnormalities in the Ts65Dn mouse model of down syndrome
Author:
Publisher
Wiley
Subject
General Neuroscience
Reference80 articles.
1. A simple dot-immunobinding assay for quantification of synaptophysin-like immunoreactivity in human brain.
2. Discovery and genetic localization of Down syndrome cerebellar phenotypes using the Ts65Dn mouse
3. Dendritic atrophy in children with Down's syndrome
4. Dual channel confocal laser scanning microscopy of lucifer yellow-microinjected human brain cells combined with Texas red immunofluorescence
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1. Hippocampal CA1 Pyramidal Neurons Display Sublayer and Circuitry Dependent Degenerative Expression Profiles in Aged Female Down Syndrome Mice;Journal of Alzheimer's Disease;2024-08-20
2. Early chronic fasudil treatment rescues hippocampal alterations in the Ts65Dn model for down syndrome;Neurochemistry International;2024-03
3. Senescent hearts from male Ts65Dn mice exhibit preserved function but altered size and nicotinamide adenine dinucleotide pathway signaling;American Journal of Physiology-Regulatory, Integrative and Comparative Physiology;2024-02-01
4. Sex-specific developmental alterations in DYRK1A expression in the brain of a Down syndrome mouse model;Neurobiology of Disease;2024-01
5. Deficits in neuronal architecture but not over-inhibition are main determinants of reduced neuronal network activity in a mouse model of overexpression of Dyrk1A;Cerebral Cortex;2023-11-23
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