CCL11 enhances excitotoxic neuronal death by producing reactive oxygen species in microglia-Reference-Cited by-同舟云学术

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CCL11 enhances excitotoxic neuronal death by producing reactive oxygen species in microglia

Published:2015-07-17 Issue:12 Volume:63 Page:2274-2284
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Parajuli Bijay¹,Horiuchi Hiroshi¹,Mizuno Tetsuya¹,Takeuchi Hideyuki¹,Suzumura Akio¹

Affiliation:

1. Department of Neuroimmunology; Research Institute of Environmental Medicine, Nagoya University; Chikusa-Ku Nagoya Japan

Funder

Grant-in-Aid for Scientific Research on Innovative Areas from the Ministry of Education, Culture, Sports, Science and Technology, Japan

Advanced Research for Medical Products Mining Program of the National Institute of Biomedical Innovation (NIBIO), Japan

Ministry of Health, Labour and Welfare of Japan

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/glia.22892/fullpdf

Reference63 articles.

1. Chemokine: receptor structure, interactions, and antagonism;Allen;Annu Rev Immunol,2007

2. Immune function of microglia;Aloisi;Glia,2001

3. RANTES promotes growth and survival of human first-trimester forebrain astrocytes;Bakhiet;Nat Cell Biol,2001

4. CNS-specific immunity at the choroid plexus shifts toward destructive Th2 inflammation in brain aging;Baruch;Proc Natl Acad Sci USA,2013

5. CCR3 blockade as a new therapy for asthma;Bertrand;Expert Opin Invest Drugs,2000

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