Imbalanced GSH/ROS and sequential cell death-Reference-Cited by-同舟云学术

Imbalanced GSH/ROS and sequential cell death

Published:2021-11-02 Issue:1 Volume:36 Page:
ISSN:1095-6670
Container-title:Journal of Biochemical and Molecular Toxicology
language:en
Short-container-title:J Biochem & Molecular Tox

Author:

Liu Ting¹,Sun Li²³,Zhang Yubin²³,Wang Yonglin¹,Zheng Jiang¹^ORCID

Affiliation:

1. State Key Laboratory of Functions and Applications of Medicinal Plants, Guizhou Provincial Key Laboratory of Pharmaceutics Guizhou Medical University Guiyang China

2. Engineering Research Center for the Development and Application of Ethnic Medicine and TCM (Ministry of Education) Guizhou Medical University Guiyang China

3. School of Pharmaceutical Sciences Guizhou Medical University Guiyang China

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Toxicology,Molecular Biology,Molecular Medicine,Biochemistry,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbt.22942

Reference94 articles.

1. Antimycin A induces death of the human pulmonary fibroblast cells via ROS increase and GSH depletion

2. The anti-cancer effect of betulinic acid in u937 human leukemia cells is mediated through ROS-dependent cell cycle arrest and apoptosis

3. 3′-Hydroxy-4′-methoxy-β-methyl-β-nitrostyrene inhibits tumor growth through ROS generation and GSH depletion in lung cancer cells

4. Tryptophan metabolite 3-hydroxyanthranilic acid selectively induces activated T cell death via intracellular GSH depletion

5. Vitamin D upregulates glutamate cysteine ligase and glutathione reductase, and GSH formation, and decreases ROS and MCP-1 and IL-8 secretion in high-glucose exposed U937 monocytes

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