Endocannabinoid activation of CB1receptors contributes to long-lasting reversal of neuropathic pain by repetitive spinal cord stimulation-Reference-Cited by-同舟云学术

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Endocannabinoid activation of CB1receptors contributes to long-lasting reversal of neuropathic pain by repetitive spinal cord stimulation

Published:2017-01-20 Issue:5 Volume:21 Page:804-814
ISSN:1090-3801
Container-title:European Journal of Pain
language:en
Short-container-title:Eur J Pain

Author:

Sun L.¹,Tai L.¹,Qiu Q.¹,Mitchell R.²,Fleetwood-Walker S.²,Joosten E.A.³,Cheung C.W.¹

Affiliation:

1. Laboratory and Clinical Research Institute for Pain; Department of Anaesthesiology; The University of Hong Kong; HKSAR China

2. Centre for Integrative Physiology; Edinburgh Medical School: Biomedical Sciences; The University of Edinburgh; UK

3. Department of Anesthesiology/Pain Management; The University Pain Center Maastricht; Maastricht University Medical Center; The Netherlands

Funder

Department of Anaesthesiology

Li Ka Shing Faculty of Medicine

University of Hong Kong

Publisher

Wiley

Subject

Anesthesiology and Pain Medicine

Reference51 articles.

1. Early endogenous activation of CB1 and CB2 receptors after spinal cord injury is a protective response involved in spontaneous recovery;Arevalo-Martin;PLoS ONE,2012

2. CB2 receptor-mediated antihyperalgesia: Possible direct involvement of neural mechanisms;Beltramo;Eur J Neurosci,2006

3. Spinal cannabinoid CB2 receptors as a target for neuropathic pain: An investigation using chronic constriction injury;Brownjohn;Neuroscience,2012

4. Dimerization with cannabinoid receptors allosterically modulates delta opioid receptor activity during neuropathic pain;Bushlin;PLoS ONE,2012

5. Pharmacological characterization of endocannabinoid transport and fatty acid amide hydrolase inhibitors;Dickason-Chesterfield;Cell Mol Neurobiol,2006

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