Molecular changes in the absence of severe pathology in the pulvinar in dementia with Lewy bodies

Author:

Erskine Daniel1,Ding Jinhui2,Thomas Alan J.1,Kaganovich Alice2,Khundakar Ahmad A.3,Hanson Peter S.4,Taylor John-Paul1,McKeith Ian G.1,Attems Johannes1,Cookson Mark R.2,Morris Christopher M.12

Affiliation:

1. Institute of Neuroscience; Newcastle University; Newcastle upon Tyne UK

2. Laboratory of Neurogenetics, National Institutes of Health; Bethesda Maryland USA

3. School of Science, Engineering and Design; Teesside University; Middlesbrough UK

4. Institute of Cellular Medicine; Newcastle University; Newcastle upon Tyne UK

Funder

NHS National Institute of Health Research Biomedical Research

Newcastle upon Tyne Hospitals NHS Foundation Trust and Newcastle University

National Institute on Aging

NIHR

NIH

Publisher

Wiley

Subject

Neurology (clinical),Neurology

Reference52 articles.

1. Is dementia with Lewy bodies the second most common cause of dementia?;Heidebrink;J Geriatr Psychiatry Neurol,2002

2. Diagnosis and management of dementia with Lewy bodies: Fourth consensus report of the DLB Consortium [published online ahead of print June 9, 2017];McKeith;Neurology

3. Alpha-synuclein in Lewy bodies;Spillantini;Nature,1997

4. Diagnosis and management of dementia with Lewy bodies: third report of the DLB Consortium;McKeith;Neurology,2005

5. Neuropathological and genetic correlates of survival and dementia onset in synucleinopathies: a retrospective analysis;Irwin;Lancet Neurol,2017

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