MKP1 overexpression reduces TNF‐α‐induced cardiac injury via suppressing mitochondrial fragmentation and inhibiting the JNK–MIEF1 pathways-Reference-Cited by-同舟云学术

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MKP1 overexpression reduces TNF‐α‐induced cardiac injury via suppressing mitochondrial fragmentation and inhibiting the JNK–MIEF1 pathways

Published:2019-02-10 Issue:9 Volume:234 Page:16148-16159
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Chang Wei¹^ORCID,Feng Min¹,Li Yuexia¹,Sun Ya²,Sun Lulu³

Affiliation:

1. Department of Intensive Care Unit The First Affiliated Hospital of Zhengzhou University Zhengzhou China

2. Department of Pharmacy The First Affiliated Hospital of Zhengzhou University Zhengzhou China

3. Department of Ultrasound The First Affiliated Hospital of Zhengzhou University Zhengzhou China

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.28273

Reference44 articles.

1. Methylglyoxal-derived advanced glycation end products contribute to negative cardiac remodeling and dysfunction post-myocardial infarction

2. Melatonin: Antioxidant and modulatory properties in age-related changes duringTrypanosoma cruziinfection

3. A new key player in VEGF-dependent angiogenesis in human hepatocellular carcinoma: dimethylarginine dimethylaminohydrolase 1

4. Transforming growth factor-β-mediated CD44/STAT3 signaling contributes to the development of atrial fibrosis and fibrillation

5. Melatonin successfully rescues hippocampal bioenergetics and improves cognitive function following drug intoxication by promoting Nrf2-ARE signaling activity

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