Alterations in driver genes are predictive of survival in patients with resected pancreatic ductal adenocarcinoma-Reference-Cited by-同舟云学术

Alterations in driver genes are predictive of survival in patients with resected pancreatic ductal adenocarcinoma

Published:2020-06-23 Issue:17 Volume:126 Page:3939-3949
ISSN:0008-543X
Container-title:Cancer
language:en
Short-container-title:Cancer

Author:

McIntyre Caitlin A.¹^ORCID,Lawrence Sharon A.¹,Richards Allison L.²,Chou Joanne F.³,Wong Winston⁴,Capanu Marinela³,Berger Michael F.²⁵,Donoghue Mark T. A.²,Yu Kenneth H.⁴⁶^ORCID,Varghese Anna M.⁴⁶,Kelsen David P.⁴⁶^ORCID,Park Wungki⁴⁶,Balachandran Vinod P.¹⁶,Kingham T. Peter¹,D’Angelica Michael I.¹,Drebin Jeffrey A.¹,Jarnagin William R.¹,Iacobuzio‐Donahue Christine A.⁵⁶⁷,Allen Peter J.¹,O’Reilly Eileen M.⁴⁶^ORCID

Affiliation:

1. Hepatopancreatobiliary Service Department of Surgery Memorial Sloan Kettering Cancer Center New York New York

2. Marie‐Josee and Henry R. Kravis Center for Molecular Oncology Memorial Sloan Kettering Cancer Center New York New York

3. Department of Biostatistics and Epidemiology Memorial Sloan Kettering Cancer Center New York New York

4. Gastrointestinal Oncology Service Department of Medicine Memorial Sloan Kettering Cancer Center New York New York

5. Department of Pathology Memorial Sloan Kettering Cancer Center New York New York

6. David M. Rubenstein Center for Pancreatic Cancer Research Memorial Sloan Kettering Cancer Center New York New York

7. Human Oncology and Pathogenesis Program Memorial Sloan Kettering Cancer Center New York New York

Funder

P30 Cancer Center Support Grant

Reiss Family Foundation

David M. Rubenstein Center for Pancreatic Cancer Research

Publisher

Wiley

Subject

Cancer Research,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/cncr.33038

Reference41 articles.

1. Projecting Cancer Incidence and Deaths to 2030: The Unexpected Burden of Thyroid, Liver, and Pancreas Cancers in the United States

2. Genomic analyses identify molecular subtypes of pancreatic cancer

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5. Whole genomes redefine the mutational landscape of pancreatic cancer

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