Hyperglycemic environment disrupts phosphate transporter function and promotes calcification processes in podocytes and isolated glomeruli-Reference-Cited by-同舟云学术

Hyperglycemic environment disrupts phosphate transporter function and promotes calcification processes in podocytes and isolated glomeruli

Published:2022-02-12 Issue:5 Volume:237 Page:2478-2491
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:Journal Cellular Physiology

Author:

Kulesza Tomasz¹^ORCID,Typiak Marlena¹²^ORCID,Rachubik Patrycja¹^ORCID,Audzeyenka Irena¹³,Rogacka Dorota¹³^ORCID,Angielski Stefan¹,Saleem Moin A.⁴,Piwkowska Agnieszka¹³^ORCID

Affiliation:

1. Laboratory of Molecular and Cellular Nephrology, Mossakowski Medical Research Institute Polish Academy of Sciences Gdansk Poland

2. Department of General and Medical Biochemistry, Faculty of Biology University of Gdansk Gdansk Poland

3. Department of Molecular Biotechnology, Faculty of Chemistry University of Gdansk Gdansk Poland

4. Bristol Renal University of Bristol Bristol UK

Funder

Narodowe Centrum Nauki

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.30700

Reference50 articles.

1. XPR1 mutations are a rare cause of primary familial brain calcification

2. Risedronate Attenuates Podocyte Injury in Phosphate Transporter-Overexpressing Rats

3. Cathepsin C is a novel mediator of podocyte and renal injury induced by hyperglycemia

4. The PKGIα–Rac1 pathway is a novel regulator of insulin‐dependent glucose uptake in cultured rat podocytes

5. Role of pyrophosphate in vascular calcification in chronic kidney disease

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