TSC-22 Promotes Interleukin-2-Deprivation Induced Apoptosis in T-Lymphocytes
Author:
Affiliation:
1. UMR 996-Inflammation, Chemokines and Immunopathology, Inserm; Univ Paris-Sud; Université Paris-Saclay; Châtenay-Malabry 92296 France
Publisher
Wiley
Subject
Cell Biology,Molecular Biology,Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/jcb.25485/fullpdf
Reference44 articles.
1. Erythropoietin-induced phosphorylation/degradation of BIM contributes to survival of erythroid cells;Abutin;Exp Hematol,2009
2. FoxO3 mediates antagonistic effects of glucocorticoids and interleukin-2 on glucocorticoid-induced leucine zipper expression;Asselin-Labat;Mol Endocrinol,2005
3. GILZ, a new target for the transcription factor FoxO3, protects T lymphocytes from interleukin-2 withdrawal-induced apoptosis;Asselin-Labat;Blood,2004
4. Glucocorticoid-induced leucine zipper (GILZ): A new important mediator of glucocorticoid action;Ayroldi;Faseb J,2009
5. Role of GILZ in immune regulation, glucocorticoid actions and rheumatoid arthritis;Beaulieu;Nat Rev Rheumatol,2011
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