Umbelliferone protects against methylglyoxal‐induced HUVECs dysfunction through suppression of apoptosis and oxidative stress-Reference-Cited by-同舟云学术

Umbelliferone protects against methylglyoxal‐induced HUVECs dysfunction through suppression of apoptosis and oxidative stress

Published:2022-10-12 Issue:4 Volume:43 Page:490-499
ISSN:0260-437X
Container-title:Journal of Applied Toxicology
language:en
Short-container-title:J of Applied Toxicology

Author:

Zhang Shunxiao¹,Zhang Sheng¹²,Li Yuan‐Yuan¹,Zhang Yan¹,Wang Hua¹,Chen Yue¹^ORCID,Sun Mingyu²³

Affiliation:

1. Department of Endocrinology, Baoshan Branch Shuguang Hospital Affiliated to Shanghai University of Traditional Chinese Medicine Shanghai China

2. Department of Endocrinology, Shuguang Hospital Affiliated to Shanghai University of Traditional Chinese Medicine Shanghai University of Traditional Chinese Medicine Shanghai China

3. Key Laboratory of Liver and Kidney Diseases, Institute of Liver Diseases Shuguang Hospital Affiliated to Shanghai University of Traditional Chinese Medicine Shanghai China

Publisher

Wiley

Subject

Toxicology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jat.4399

Reference50 articles.

1. Lectin-like oxidized low-density lipoprotein receptor-1 (LOX-1): a crucial driver of atherosclerotic cardiovascular disease

2. Nephroprotective effect of umbelliferone against cisplatin‐induced kidney damage is mediated by regulation of NRF2, cytoglobin, SIRT1/FOXO‐3, and NF‐ k B‐p65 signaling pathways

Umbelliferone Inhibits Spermatogenic Defects and Testicular Injury in Lead-Intoxicated Rats by Suppressing Oxidative Stress and Inflammation, and Improving Nrf2/HO-1 Signaling

4. Umbelliferone prevents isoproterenol-induced myocardial injury by upregulating Nrf2/HO-1 signaling, and attenuating oxidative stress, inflammation, and cell death in rats

5. The use of human umbilical vein endothelial cells (HUVECs) as an in vitro model to assess the toxicity of nanoparticles to endothelium: a review

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