TP53 and p16INK4A, but not H-KI-Ras, are involved in tumorigenesis and progression of pleomorphic adenomas-Reference-Cited by-同舟云学术

TP53 and p16INK4A, but not H-KI-Ras, are involved in tumorigenesis and progression of pleomorphic adenomas

Published:2006 Issue:3 Volume:207 Page:654-659
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J. Cell. Physiol.

Author:

Augello Claudia,Gregorio Valter,Bazan Viviana,Cammareri Patrizia,Agnese Valentina,Cascio Sandra,Corsale Simona,Calò Valentina,Gullo Arianna,Passantino Rita,Gargano Grazia,Bruno Loredana,Rinaldi Gaetana,Morello Vincenza,Gerbino Aldo,Tomasino Rosa Maria,Macaluso Marcella,Surmacz Eva,Russo Antonio

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Reference40 articles.

1. Frequent mutations ofCDKN2 in primary pancreatic adenocarcinomas

2. Specific codon 13 K-ras mutations are predictive of clinical outcome in colorectal cancer patients, whereas codon 12 K-ras mutations are associated with mucinous histotype

3. Specific TP53 and/or Ki-ras mutations as independent predictors of clinical outcome in sporadic colorectal adenocarcinomas: results of a 5-year Gruppo Oncologico dell'Italia Meridionale (GOIM) prospective study

4. Laser pressure catapulting (LPC): Optimization LPC-system and genotyping of colorectal carcinomas

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