Ablation of Proliferating Osteoblast Lineage Cells After Fracture Leads to Atrophic Nonunion in a Mouse Model

Author:

Hixon Katherine R.1ORCID,McKenzie Jennifer A.1,Sykes David A.W.2ORCID,Yoneda Susumu1,Hensley Austin13,Buettmann Evan G.13ORCID,Zheng Hongjun1,Skouteris Dimitrios1,McAlinden Audrey145ORCID,Miller Anna N.1ORCID,Silva Matthew J.13ORCID

Affiliation:

1. Department of Orthopaedic Surgery Washington University in St. Louis St. Louis MO USA

2. Department of Biology Washington University in St. Louis St. Louis MO USA

3. Department of Biomedical Engineering Washington University in St. Louis St. Louis MO USA

4. Department of Cell Biology & Physiology Washington University in St. Louis St. Louis MO USA

5. St. Louis Shriners Hospital Research Center Shriners Hospital for Children St. Louis MO USA

Funder

National Institute of Arthritis and Musculoskeletal and Skin Diseases

University of Arkansas for Medical Sciences

Washington University in St. Louis

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Reference54 articles.

1. Cellular and molecular characterization of a murine non-union model

2. Fracture non‐union epidemiology and treatment;Nandra R;Dent Traumatol,2016

3. Enhancement of fracture-healing.

4. Proximal humerus and humeral shaft nonunions;Cadet ER;J Am Acad Orthop Surg,2013

5. The relative incidence of fracture non-union in the Scottish population (5.17 million): a 5-year epidemiological study

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