S-adenosylmethionine inhibits lipopolysaccharide-induced gene expression via modulation of histone methylation

Author:

Ara Ainhoa Iglesias,Xia Meng,Ramani Komal,Mato José M.,Lu Shelly C.

Funder

National Institutes of Health

Plan Nacional of I+D SAF

Cell Culture Core of the University of Southern California Research Center for Liver Diseases

Postdoctoral fellowship supported by the Centro de Investigación Cooperativa en Biosciencías

Postdoctoral fellowship of the Training Program in Alcoholic Liver and Pancreatic Diseases

Publisher

Wiley

Subject

Hepatology

Reference38 articles.

1. Role of S-adenosyl-L-methionine in liver health and injury;Mato;HEPATOLOGY,2007

2. Lipopolysaccharides in liver injury: molecular mechanisms of Kupffer cell activation;Su;Am J Physiol Gastrointest Liver Physiol,2002

3. Tumor necrosis factor: a pleiotropic cytokine and therapeutic target;Tracey;Annu Rev Med,1994

4. Nitric oxide. IV. Determinants of nitric oxide protection and toxicity in liver;Li;Am J Physiol,1999

5. Inhibition of lipopolysaccharide-stimulated TNF-alpha promoter activity by S-adenosylmethionine and 5′-methylthioadenosine;Veal;Am J Physiol Gastrointest Liver Physiol,2004

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