Exposure of Pulmonary Artery Endothelial Cells to Nitrogen Dioxide Activates Phospholipase A1
Author:
Publisher
Wiley
Subject
Toxicology
Reference11 articles.
1. Effect of nitrogen dioxide on surface membrane fluidity and insulin receptor binding of pulmonary endothelial cells
2. A new role for phospholipase A2: protection of membranes from lipid peroxidation damage
3. Phosphatidylcholine metabolism in endothelial cells: Evidence for phospholipase A and a novel Ca2+-independent phospholipase C
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1. Long-term Air Pollution Exposure, Genome-wide DNA Methylation and Lung Function in the LifeLines Cohort Study;Environmental Health Perspectives;2018-02
2. Short-term NO2exposure is associated with long-chain fatty acids in prospective cohorts from Augsburg, Germany: results from an analysis of 138 metabolites and three exposures;International Journal of Epidemiology;2016-10
3. Plasma membrane-specific phospholipase A1 activation by nitrogen dioxide in pulmonary artery endothelial cells;Toxicology and Applied Pharmacology;1991-03
4. OXIDANT-INDUCED MECHANISM OF PLASMA MEMBRANE PHOSPHOLIPASE A1 ACTIVATION IN PULMONARY ARTERY ENDOTHELIAL CELLS;Oxidative Damage & Repair;1991
5. Oxidant injury increases cell surface receptor binding of angiotensin ii to pulmonary artery endothelial cells;Journal of Biochemical Toxicology;1990
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