Displacing hexokinase from mitochondrial voltage-dependent anion channel impairs GLT-1-mediated glutamate uptake but does not disrupt interactions between GLT-1 and mitochondrial proteins-Reference-Cited by-同舟云学术

Displacing hexokinase from mitochondrial voltage-dependent anion channel impairs GLT-1-mediated glutamate uptake but does not disrupt interactions between GLT-1 and mitochondrial proteins

Published:2014-12-26 Issue:7 Volume:93 Page:999-1008
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:Journal of Neuroscience Research

Author:

Jackson Joshua G.¹²,O'Donnell John C.¹³,Krizman Elizabeth¹,Robinson Michael B.¹²³

Affiliation:

1. Children's Hospital of Philadelphia Research Institute; Philadelphia Pennsylvania

2. Department of Pediatrics; University of Pennsylvania; Philadelphia Pennsylvania

3. Department of Pharmacology; University of Pennsylvania; Philadelphia Pennsylvania

Funder

NINDS

NIH

The Institutional Intellectual and Developmental Disabilities Research Center

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/jnr.23533/fullpdf

Reference63 articles.

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3. Functional comparisons of three glutamate transporter subtypes cloned from human motor cortex;Arriza;J Neurosci,1994

4. The glutamate transporter GLAST participates in a macromolecular complex that supports glutamate metabolism;Bauer;Neurochem Int,2012

5. Hexokinase of rat brain mitochondria: relative importance of adenylate kinase and oxidative phosphorylation as sources of substrate ATP and interaction with intramitochondrial compartments of ATP and ADP;BeltrandelRio;Arch Biochem Biophys,1991

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