Comment on developmental toxicity evaluation on vorinostat and relationship with HDAC inhibition-Reference-Cited by-同舟云学术

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Comment on developmental toxicity evaluation on vorinostat and relationship with HDAC inhibition

Published:2007 Issue:5 Volume:80 Page:417-418
ISSN:1542-9733
Container-title:Birth Defects Research Part B: Developmental and Reproductive Toxicology
language:en
Short-container-title:Birth Defect Res B

Author:

Giavini Erminio

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Developmental Biology,Toxicology,Embryology

Reference5 articles.

1. Boric acid inhibits embryonic histone deacetylases: A suggested mechanism to explain boric acid-related teratogenicity

2. , . 2007b. Relationship between embryonic histonic hypercatylation and axial skeletal defects in mouse exposed to the three HDAC inhibitors apicidin, MS-275, sodium butyrate. Toxicol Sciences (published online, doi: 10.1093/toxsci/kfm115).

3. Comparative study of sodium valproate-induced skeletal malformations using single or double staining methods

4. Inhibition of histone deacetylase activity on specific embryonic tissues as a new mechanism for teratogenicity

5. Assessment of developmental toxicity of vorinostat, a histone deacetylase inhibitor, in Sprague-Dawley rats and Dutch Belted rabbits

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Why Hydroxamates May Not Be the Best Histone Deacetylase Inhibitors-What Some May Have Forgotten or Would Rather Forget?;ChemMedChem;2015-11-25

2. Toxicological and metabolic considerations for histone deacetylase inhibitors;Expert Opinion on Drug Metabolism & Toxicology;2013-01-04

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