CaMKII: Therapeutic target in vascular diseases warranted for Clinical and Translational Discovery?-Reference-Cited by-同舟云学术

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CaMKII: Therapeutic target in vascular diseases warranted for Clinical and Translational Discovery?

Published:2022-07-24 Issue:3 Volume:2 Page:
ISSN:2768-0622
Container-title:Clinical and Translational Discovery
language:en
Short-container-title:Clinical and Translational Dis

Author:

Zhang Mao¹²,Lam Chi Keung³^ORCID

Affiliation:

1. Stanford Cardiovascular Institute Stanford University School of Medicine Stanford California USA

2. Department of Medicine Division of Cardiovascular Medicine Stanford University School of Medicine Stanford California USA

3. Department of Biological Sciences University of Delaware Newark Delaware USA

Publisher

Wiley

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ctd2.119

Reference12 articles.

1. Calcium/calmodulin-dependent kinase 2 mediates Epac-induced spontaneous transient outward currents in rat vascular smooth muscle

2. Differential Control of Calcium Homeostasis and Vascular Reactivity by Ca 2+ /Calmodulin-Dependent Kinase II

3. Calcium in Vascular Smooth Muscle Cell Elasticity and Adhesion: Novel Insights Into the Mechanism of Action

4. Calcium/Calmodulin‐Dependent Kinase II Inhibition in Smooth Muscle Reduces Angiotensin II–Induced Hypertension by Controlling Aortic Remodeling and Baroreceptor Function

5. Camk2n1 Is a Negative Regulator of Blood Pressure, Left Ventricular Mass, Insulin Sensitivity, and Promotes Adiposity

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Role of CaMKII in diabetes induced vascular injury and its interaction with anti-diabetes therapy;Reviews in Endocrine and Metabolic Disorders;2023-12-08

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