Overexpression of TRIM32 promotes pancreatic β‐cell autophagic cell death through Akt/mTOR pathway under high glucose conditions-Reference-Cited by-同舟云学术

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Overexpression of TRIM32 promotes pancreatic β‐cell autophagic cell death through Akt/mTOR pathway under high glucose conditions

Published:2022-08-30 Issue:12 Volume:46 Page:2095-2106
ISSN:1065-6995
Container-title:Cell Biology International
language:en
Short-container-title:Cell Biology International

Author:

Wan Tingting¹²,Wang Yidan¹²,Wang Chunxu²³,Wang Hongjie¹,Li Xiudan¹²,Li Yanbo¹^ORCID

Affiliation:

1. Department of Endocrinology The First Affiliated Hospital of Harbin Medical University Harbin Heilongjiang China

2. The Key Laboratory of Myocardial Ischemia Ministry of Education Harbin Heilongjiang China

3. Department of Hematology The First Affiliated Hospital of Harbin Medical University Harbin Heilongjiang China

Funder

Natural Science Foundation of Guangdong Province

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/cbin.11897

Reference39 articles.

1. mTORC1 Signaling: A Double-Edged Sword in Diabetic β Cells

2. Autophagy plays a protective role in endoplasmic reticulum stress-mediated pancreatic β cell death

3. Pancreatic β-Cell Failure Mediated by mTORC1 Hyperactivity and Autophagic Impairment

4. The double-edged effect of autophagy in pancreatic beta cells and diabetes

5. Trim32 reduces PI3K–Akt–FoxO signaling in muscle atrophy by promoting plakoglobin–PI3K dissociation

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1. Adipose stem cell‑derived exosomes promote high glucose-induced wound healing by regulating the TRIM32/STING axis;Archives of Dermatological Research;2024-06-01

2. Transcriptional co-activators: emerging roles in signaling pathways and potential therapeutic targets for diseases;Signal Transduction and Targeted Therapy;2023-11-13

3. TRIM32 promotes oral squamous cell carcinoma progression by enhancing FBP2 ubiquitination and degradation;Biochemical and Biophysical Research Communications;2023-10

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