<scp>ThPOK</scp> Inhibits Osteoclast Formation Via <scp>NFATc1</scp> Transcription and Function-Reference-Cited by-同舟云学术

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ThPOK Inhibits Osteoclast Formation Via NFATc1 Transcription and Function

Published:2022-03-14 Issue:4 Volume:6 Page:
ISSN:2473-4039
Container-title:JBMR Plus
language:en
Short-container-title:JBMR Plus

Author:

Zou Wei¹^ORCID,Izawa Takashi¹²,Rohatgi Nidhi¹,Zou Steven Y¹,Li Yongjia¹³,Teitelbaum Steven L¹⁴^ORCID

Affiliation:

1. Division of Anatomic and Molecular Pathology, Department of Pathology and Immunology Washington University School of Medicine St. Louis MO USA

2. Department of Orthodontics Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University Okayama Japan

3. Department of Pharmacology Jiangsu University School of Medicine Zhenjiang China

4. Division of Bone and Mineral Diseases, Department of Medicine Washington University School of Medicine St. Louis MO USA

Funder

Foundation for Barnes-Jewish Hospital

National Institutes of Health

School of Medicine

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbm4.10613

Reference23 articles.

1. alphavbeta3 and macrophage colony-stimulating factor: partners in osteoclast biology

2. CSF-1 regulation of the wandering macrophage: complexity in action

3. RANK-mediated amplification of TRAF6 signaling leads to NFATc1 induction during osteoclastogenesis

4. Role of RANKL in physiological and pathological bone resorption and therapeutics targeting the RANKL-RANK signaling system

5. JNK1 modulates osteoclastogenesis through both c-Jun phosphorylation-dependent and -independent mechanisms

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