Identification of the breakpoints at 1p36.2 and 3p21.3 in an AML(M3) patient who had t(1;3)(p36.2;p21.3) at the third relapse-Reference-Cited by-同舟云学术

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Identification of the breakpoints at 1p36.2 and 3p21.3 in an AML(M3) patient who had t(1;3)(p36.2;p21.3) at the third relapse

Published:2002-10-11 Issue:4 Volume:35 Page:365-367
ISSN:1045-2257
Container-title:Genes, Chromosomes and Cancer
language:en
Short-container-title:Genes Chromosom. Cancer

Author:

Tri Nguyen Khanh,Xinh Phan Thi,Nagao Hiromasa,Izumi Tohru,Ozawa Keiya,Toyoda Atsushi,Hattori Masahira,Sakaki Yoshiyuki,Tokunaga Katsushi,Sato Yuko

Publisher

Wiley

Subject

Cancer Research,Genetics

Reference5 articles.

1. A novel gene, MEL1, mapped to 1p36.3 is highly homologous to the MDS1/EVI1 gene and is transcriptionally activated in t(1;3)(p36;q21)-positive leukemia cells

2. Novel SH3 protein encoded by the AF3p21 gene is fused to the mixed lineage leukemia protein in a therapy-related leukemia with t(3;11) (p21;q23)

3. Second relapse of acute promyelocytic leukemia (ANLL-M3) with t(15;17) and t(1;3)(p36;q21)

4. t(1;3)(p36;p21) is a recurring therapy-related translocation

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