Salbutamol repurposing ameliorates neuromuscular junction defects and muscle atrophy in <i>Col6a1<sup>−/−</sup></i> mouse model of collagen VI‐related myopathies-Reference-Cited by-同舟云学术

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Salbutamol repurposing ameliorates neuromuscular junction defects and muscle atrophy in Col6a1^−/− mouse model of collagen VI‐related myopathies

Published:2024-07 Issue:7 Volume:14 Page:
ISSN:2001-1326
Container-title:Clinical and Translational Medicine
language:en
Short-container-title:Clinical & Translational Med

Author:

Calabrò Sonia¹²,Nogara Leonardo³⁴⁵,Jian Yongzhi⁶,Valentin Manuel⁶,Bizzotto Dario¹,Braghetta Paola¹,Russo Loris¹,Gambarotto Lisa¹,Blaauw Bert³⁴,Hashemolhosseini Said⁶,Bonaldo Paolo¹,Cescon Matilde¹^ORCID

Affiliation:

1. Department of Molecular Medicine University of Padova Padova Italy

2. Department of Biology University of Padova Padova Italy

3. Department of Biomedical Sciences University of Padova Padova Italy

4. Venetian Institute of Molecular Medicine Padova Italy

5. Department of Pharmaceutical and Pharmacological Sciences University of Padova Padova Italy

6. Institut für Biochemie Friedrich‐Alexander‐Universität Erlangen‐Nürnberg Erlangen Germany

Funder

Università degli Studi di Padova

Fondazione Telethon

Publisher

Wiley

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ctm2.1688

Reference10 articles.

1. Collagen VI deficiency induces early onset myopathy in the mouse: an animal model for Bethlem myopathy

2. Cyclosporin A corrects mitochondrial dysfunction and muscle apoptosis in patients with collagen VI myopathies

3. Reactivation of autophagy by spermidine ameliorates the myopathic defects of collagen VI-null mice

4. Sustained oral spermidine supplementation rescues functional and structural defects in COL6-deficient myopathic mice

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