TRPC3‐mediated Ca 2+ signals as a promising strategy to boost therapeutic angiogenesis in failing hearts: The role of autologous endothelial colony forming cells-Reference-Cited by-同舟云学术

TRPC3‐mediated Ca 2+ signals as a promising strategy to boost therapeutic angiogenesis in failing hearts: The role of autologous endothelial colony forming cells

Published:2017-09-27 Issue:5 Volume:233 Page:3901-3917
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Moccia Francesco¹^ORCID,Lucariello Angela²,Guerra Germano³

Affiliation:

1. Laboratory of General Physiology, Department of Biology and Biotechnology “Lazzaro Spallanzani”University of PaviaPaviaItaly

2. Department of Mental and Physical Health and Preventive Medicine, Section of Human AnatomyUniversy of Campania “L. Vanvitelli”NaplesItaly

3. Department of Medicine and Health Sciences “Vincenzo Tiberio”University of MoliseCampobassoItaly

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.26152

Reference206 articles.

1. Stim1 and Orai1 Mediate CRAC Currents and Store-Operated Calcium Entry Important for Endothelial Cell Proliferation

2. Mobilizing Endothelial Progenitor Cells

3. Myocardial regeneration of the failing heart

4. Pharmacological evidence for a role of the transient receptor potential canonical 3 (TRPC3) channel in endoplasmic reticulum stress-induced apoptosis of human coronary artery endothelial cells

5. Ca2+ Influx through Reverse Mode Na+/Ca2+ Exchange Is Critical for Vascular Endothelial Growth Factor-mediated Extracellular Signal-regulated Kinase (ERK) 1/2 Activation and Angiogenic Functions of Human Endothelial Cells

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