c‐Jun N‐Terminal Kinases (JNKs) Are Critical Mediators of Osteoblast Activity In Vivo-Reference-Cited by-同舟云学术

c‐Jun N‐Terminal Kinases (JNKs) Are Critical Mediators of Osteoblast Activity In Vivo

Published:2017-08-14 Issue:9 Volume:32 Page:1811-1815
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Xu Ren¹,Zhang Chao²³,Shin Dong Yeon¹,Kim Jung‐Min⁴,Lalani Sarfaraz¹,Li Na¹,Yang Yeon‐Suk⁴,Liu Yifang¹,Eiseman Mark¹,Davis Roger J⁵,Shim Jae‐Hyuck⁴,Greenblatt Matthew B¹

Affiliation:

1. Department of Pathology and Laboratory MedicineWeill Cornell Medical CollegeCornell UniversityNew YorkNYUSA

2. Institute for Computational BiomedicineWeill Cornell Medical CollegeCornell UniversityNew YorkNYUSA

3. Department of MedicineDivision of Hematology and Medical OncologyWeill Cornell Medical CollegeCornell UniversityNew YorkNYUSA

4. Division of RheumatologyDepartment of MedicineUniversity of Massachusetts Medical SchoolWorcesterMAUSA

5. Program in Molecular MedicineUniversity of Massachusetts Medical SchoolWorcesterMAUSA

Funder

Burroughs Wellcome Fund

National Institutes of Health

U.S. Department of Defense

National Institute of Arthritis and Musculoskeletal and Skin Diseases

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbmr.3184

Reference24 articles.

1. The stress-activated protein kinase subfamily of c-Jun kinases

2. Identification of an oncoprotein- and UV-responsive protein kinase that binds and potentiates the c-Jun activation domain.

3. Selective interaction of JNK protein kinase isoforms with transcription factors.

4. JNK1: A protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain

5. The JNK signal transduction pathway

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