Generation ofPex5-loxP mice allowing the conditional elimination of peroxisomes
Author:
Publisher
Wiley
Subject
Cell Biology,Endocrinology,Genetics
Reference7 articles.
1. A mouse model for Zellweger syndrome
2. Mitochondrial Alterations Caused by Defective Peroxisomal Biogenesis in a Mouse Model for Zellweger Syndrome (PEX5 Knockout Mouse)
3. Structure-Function Analyses of Thrombomodulin by Gene-Targeting in Mice: The Cytoplasmic Domain Is Not Required for Normal Fetal Development
4. Peroxisome biogenesis disorders
5. Protamine-Cre recombinase transgenes efficiently recombine target sequences in the male germ line of mice, but not in embryonic stem cells
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1. Loss of pex5 sensitizes zebrafish to fasting due to deregulated mitochondria, mTOR, and autophagy;Cellular and Molecular Life Sciences;2023-02-23
2. Normal plasmalogen levels are maintained in tissues from mice with hepatocyte-specific deletion in peroxin 5;Brain Research Bulletin;2023-02
3. A Pex7 Deficient Mouse Series Correlates Biochemical and Neurobehavioral Markers to Genotype Severity—Implications for the Disease Spectrum of Rhizomelic Chondrodysplasia Punctata Type 1;Frontiers in Cell and Developmental Biology;2022-07-11
4. Peroxisomes Are Critical for the Development and Maintenance of B1 and Marginal Zone B Cells but Dispensable for Follicular B Cells and T Cells;The Journal of Immunology;2022-02-15
5. Cell Type-Selective Loss of Peroxisomal β-Oxidation Impairs Bipolar Cell but Not Photoreceptor Survival in the Retina;Cells;2022-01-04
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