Conditional inactivation of the peroxisome biogenesisPex13 gene by Cre-loxP excision
Author:
Publisher
Wiley
Subject
Cell Biology,Endocrinology,Genetics
Reference5 articles.
1. PEX13 Is Mutated in Complementation Group 13 of the Peroxisome-Biogenesis Disorders
2. High-efficiency deleter mice show that FLPe is an alternative to Cre-loxP
3. Acre-transgenic mouse strain for the ubiquitous deletion ofloxP-flanked gene segments including deletion in germ cells
4. Nonsense and temperature-sensitive mutations in PEX13 are the cause of complementation group H of peroxisome biogenesis disorders
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1. A Mouse Model System to Study Peroxisomal Roles in Neurodegeneration of Peroxisome Biogenesis Disorders;Advances in Experimental Medicine and Biology;2020
2. Peroxisomes in cardiomyocytes and the peroxisome / peroxisome proliferator-activated receptor-loop;Thrombosis and Haemostasis;2015-05
3. Mouse Models with Peroxisome Biogenesis Defects;Molecular Machines Involved in Peroxisome Biogenesis and Maintenance;2014
4. PEX13 deficiency in mouse brain as a model of Zellweger syndrome: abnormal cerebellum formation, reactive gliosis and oxidative stress;Disease Models & Mechanisms;2011-01-01
5. Peroxisomes and peroxisomal disorders: The main facts;Experimental and Toxicologic Pathology;2010-11
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