Targeting RIPK1 in AML cells carrying FLT3‐ITD-Reference-Cited by-同舟云学术

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Targeting RIPK1 in AML cells carrying FLT3‐ITD

Published:2019-03-23 Issue:6 Volume:145 Page:1558-1569
ISSN:0020-7136
Container-title:International Journal of Cancer
language:en
Short-container-title:Int. J. Cancer

Author:

Hillert Laura K.¹,Bettermann‐Bethge Kira¹,Nimmagadda Subbaiah Chary²,Fischer Thomas²,Naumann Michael³,Lavrik Inna N.¹^ORCID

Affiliation:

1. Translational Inflammation Research, Health Campus Immunology, Infectiology and Inflammation, Center of Dynamic Systems, Medical FacultyOtto von Guericke University Magdeburg Germany

2. Department of Hematology and Oncology, Health Campus Immunology, Infectiology and InflammationOtto von Guericke University, Medical Faculty Magdeburg Germany

3. Institute of Experimental Internal Medicine, Health Campus Immunology, Infectiology and InflammationOtto von Guericke University, Medical Faculty Magdeburg Germany

Funder

Deutsche Forschungsgemeinschaft

European Regional Development Fund

Publisher

Wiley

Subject

Cancer Research,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ijc.32246

Reference50 articles.

1. Extracellular assembly and activation principles of oncogenic class III receptor tyrosine kinases

2. FLT3 Mutations: Biology and Treatment

3. Survey of Activated FLT3 Signaling in Leukemia

4. FLT3 tyrosine kinase domain mutations are biologically distinct from and have a significantly more favorable prognosis than FLT3 internal tandem duplications in patients with acute myeloid leukemia

5. The role of FLT3 in haematopoietic malignancies

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