A high‐resolution structure of the EF‐hand domain of human polycystin‐2-Reference-Cited by-同舟云学术

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A high‐resolution structure of the EF‐hand domain of human polycystin‐2

Published:2014-07-22 Issue:9 Volume:23 Page:1301-1308
ISSN:0961-8368
Container-title:Protein Science
language:en
Short-container-title:Protein Science

Author:

Allen Mark D.¹,Qamar Seema²,Vadivelu Murali K.¹,Sandford Richard N.³,Bycroft Mark¹

Affiliation:

1. MRC Laboratory of Molecular BiologyHills RoadCambridgeCB2 0QH United Kingdom

2. Department of Clinical Neurosciences, Cambridge Institute for Medical ResearchUniversity of CambridgeCambridgeCB2 0XY United Kingdom

3. Academic Department of Medical Genetics, School of Clinical MedicineUniversity of CambridgeCambridgeCB2 0QQ United Kingdom

Funder

Medical Research Council

Publisher

Wiley

Subject

Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/pro.2513

Reference27 articles.

1. Autosomal dominant polycystic kidney disease (ADPKD, MIM 173900, PKD1 and PKD2 genes, protein products known as polycystin-1 and polycystin-2)

2. PKD1 Induces p21waf1 and Regulation of the Cell Cycle via Direct Activation of the JAK-STAT Signaling Pathway in a Process Requiring PKD2

3. PKD1 interacts with PKD2 through a probable coiled-coil domain

4. PKD2 , a Gene for Polycystic Kidney Disease That Encodes an Integral Membrane Protein

5. TRP channels and kidney disease: lessons from polycystic kidney disease

Cited by 17 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The diverse effects of pathogenic point mutations on ion channel activity of a gain-of-function polycystin-2;Journal of Biological Chemistry;2023-05

2. Polycystin Channel Complexes;Annual Review of Physiology;2023-02-10

3. Post-translational modifications of the polycystin proteins;Cellular Signalling;2020-08

4. Structure and function of polycystin channels in primary cilia;Cellular Signalling;2020-08

5. Disrupting Polycystin-2 EF hand Ca2+ affinity does not alter channel function or contribute to polycystic kidney disease;Journal of Cell Science;2020-01-01

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