Sodium channel slow inactivation as a therapeutic target for myotonia congenita
Author:
Affiliation:
1. Department of Neuroscience; Cell Biology, and Physiology, Wright State University; Dayton OH
2. Department of Physiology; Emory University School of Medicine; Atlanta GA
Funder
National Institutes of Health
Publisher
Wiley
Subject
Clinical Neurology,Neurology
Reference51 articles.
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3. On the repetitive discharge in myotonic muscle fibres;Adrian;J Physiol,1974
4. Characteristics of the chloride conductance in muscle fibers of the rat diaphragm;Palade;J Gen Physiol,1977
5. Clinical and electrophysiological observations in patients with myotonic muscle disease and the therapeutic effect of N-propyl-ajmalin;Birnberger;J Neurol,1975
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