UV-sensitive rodent mutant cell lines of complementation groups 6 and 8 differ phenotypically from their human counterparts
Author:
Publisher
Wiley
Subject
Health, Toxicology and Mutagenesis,Genetics (clinical),Epidemiology
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1. Cockayne's Syndrome Fibroblasts Have Increased Sensitivity to Ultraviolet Light But Normal Rates of Unscheduled DNA Synthesis
2. Evidence for detective repair of cyclobutane pyrimidine dimers with normal repair of other DNA photoproducts in a transcriptionally active gene transfected into Cockayne syndrome cells
3. DNA repair in an active gene: Removal of pyrimidine dimers from the DHFR gene of CHO cells is much more efficient than in the genome overall
4. Complementation group assignments of moderately UV-sensitive CHO mutants isolated by large-scale screening (FAECB)
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