AH receptor, ARNT, glucocorticoid receptor, EGF receptor, EGF, TGFα, TGFβ1, TGFβ2, and TGFβ3 expression in human embryonic palate, and effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)
Author:
Publisher
Wiley
Subject
Health, Toxicology and Mutagenesis,Developmental Biology,Toxicology,Embryology
Reference42 articles.
1. (1997) Developmental toxicity of dioxin: Searching for the cellular and molecular basis of morphological responses. In: Handbook of Experimental Pharmacology 124/11: Drug Toxicity in Embryonic Development II. and , eds. New York: Springer-Verlag, pp. 407–433.
2. Retinoic acid-induced alterations in the expression of growth factors in embryonic mouse palatal shelves
3. TCDD-induced altered expression of growth factors may have a role in producing cleft palate and enhancing the incidence of clefts after coadministration of retinoic acid and TCDD
4. TCDD exposure of human embryonic palatal shelves in organ culture alters the differentiation of medial epithelial cells
5. Embryonic palatal responses to teratogens in serum-free organ culture
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