Fisher Syndrome with tetraparesis and antibody to GQ1b: Evidence for motor nerve terminal block
Author:
Affiliation:
1. Center for Neuromuscular Diseases, University “G. d'Annunzio,” Clinica Neurologica Ospedale Clinicizzato “SS Annunziata,” Via dei Vestini, I‐66013 Chieti, Italy
Publisher
Wiley
Subject
Physiology (medical),Cellular and Molecular Neuroscience,Neurology (clinical),Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/%28SICI%291097-4598%28199905%2922%3A5%3C640%3A%3AAID-MUS14%3E3.0.CO%3B2-%23
Reference18 articles.
1. The Miller Fisher syndrome. Review of the literature;Berlit P;J Clin Neuroophthalmol,1992
2. Immunoglobulin G from a patient with Miller-Fisher syndrome rapidly and reversibly depresses evoked quantal release at the neuromuscular junction of mice
3. Ganglioside composition of the human cranial nerves, with special reference to pathophysiology of Miller Fisher syndrome
4. Serum anti-GQ1b IgG antibody is associated with ophthalmoplegia in Miller Fisher syndrome and Guillain-Barre syndrome: Clinical and immunohistochemical studies
5. Serum IgG antibody to ganglioside GQ1b is a possible marker of Miller Fisher syndrome
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