Expression analysis of endogenous menin, the product of the multiple endocrine neoplasia type 1 gene, in cell lines and human tissues
Author:
Publisher
Wiley
Subject
Cancer Research,Oncology
Reference22 articles.
1. Germline mutations of the MEN1 gene in familial multiple endocrine neoplasia type 1 and related states
2. Menin Interacts with the AP1 Transcription Factor JunD and Represses JunD-Activated Transcription
3. Characterization of Mutations in Patients with Multiple Endocrine Neoplasia Type 1
4. Positional Cloning of the Gene for Multiple Endocrine Neoplasia-Type 1
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1. Loss of MEN1 leads to renal fibrosis and decreases HGF‐Adamts5 pathway activity via an epigenetic mechanism;Clinical and Translational Medicine;2022-08
2. The link between menin and pleiotrophin in the tumor biology of pancreatic neuroendocrine neoplasms;Cancer Science;2022-03-08
3. Exploring the MEN1 dependent modulation of caspase 8 and caspase 3 in human pancreatic and murine embryo fibroblast cells;Apoptosis;2021-12-08
4. A Novel Isogenic Human Cell-Based System for MEN1 Syndrome Generated by CRISPR/Cas9 Genome Editing;International Journal of Molecular Sciences;2021-11-08
5. Pancreatic Neuroendocrine Tumors: Molecular Mechanisms and Therapeutic Targets;Cancers;2021-10-12
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